叶山东
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个人简历
主要学习、工作、出国经历198707 – 199009: 内科住院医生,安徽省立医院199512 – 199712: 内分泌专业主治医师,安徽省立医院199712 – 200212: 内分泌专业副主任医师,安徽省立医院200212 – 201803: 内分泌专业主任医师,安徽省立医院198209 – 198707: 学士,皖南医学院199009 – 199307: 硕士,上海医科大学199309 – 199512: 博士,上海医科大学研究领域
糖尿病及其慢性并发症"目前正在承担的科研项目(仅限于主要负责人)项目名称、来源、经费指标卡号 总经费(万元) 起止日期胰岛素强化治疗2型糖尿病的临床研究(中央引导地方科技创新专项)财政拨款50万医院配套50万201707至202007P38MAPK介导的肾小球系膜细胞氧化应激及二甲双胍的抑制作用安徽省科技厅 财政拨款8万医院配套8万 201507至201706"近期论文
MiR-181c restrains nitration stress of endothelial cells in diabetic db/db mice through inhibiting the expression of FoxO1. Biochem Biophys Res Commun 3/3 是 (2017)Metformin ameliorates podocyte damage by restoring renal tissue nephrin expression in type 2 diabetic rats Journal of Diabetes 6/6 是 (2017)Comparison of Antidiabetic Medications during the Treatment of Atherosclerosis in T2DM Patients Mediators of Inflammation 4/4 是 (2017)Effects of metformin on blood and urine pro-inflammatory mediators in patients with type 2 diabetes Journal of Inflammation 3/3 是 (2016)Subject-driven titration of biphasic insulin aspart 30 twice daily is non-inferior to investigator-driven titration in Chinese patients with type 2 diabetes inadequately controlled with premixed human insulin: A randomiz J Diabetes Invest 6/15 否 (2016)Metformin Ameliorates Podocyte Damage by Restoring Renal Tissue Podocalyxin Expression in Type 2 Diabetic Rats Journal of Diabetes Research 5/5 是 (2015)The alpha-lipoic acid dereases urinary podocalyxin excretion in type 2 diabetics by inhibiting oxidative stress in vivo Journal of Diabetes and Its Complications 2/4 是 (2015)Relationship between Serum 25-Hydroxyvitamin D and Lower Extremity Arterial Disease in Type 2 Diabetes Mellitus Patients and the Analysis of the Intervention of Vitamin D Journal of Diabetes Research 2/2 是 (2015)Abnormal Methylation Status of the GNAS Exon 1A Region in Pseudohypohyperpara -thyroidism Combined With Turner Syndrom The American Journal of the Medical Sciences 9/9 是 (2015)Metformin inhibits nuclear factor-κB activation and inflammatory cytokines expression induced by high glucose via adenosine monophosphate-activated protein kinase activation in rat glomerular mesangial cells in vitro Chinese Medical Journal 2/5 是 (2014)内分泌科科主任;中华医学会内分泌学分会委员、安徽省医学会内分泌学分会主任委员、安徽省医学会糖尿病学分会副主任委员;安徽省临床检验中心副主任 相关热点
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