陈政
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实验室主要从事炎症与代谢类疾病分子机制研究,已在国际著名学术期刊发表SCI论文30余篇,其中通讯作者SCI论文11篇,被引800余次。陈政博士2009年毕业于中国科学院生物物理研究所获得博士学位,随后在美国密歇根大学医学院分子整合生理系从事研究工作,2015-2018年受聘东北师范大学生命科学学院教授。陈政博士于2018年加入哈尔滨工业大学生命科学中心/生命科学技术学院。招生信息博士招生: 每年招收博士1名硕士招生: 每年招收硕士2-3名研究领域
1)研究NIK信号通路在炎症和代谢类疾病中的作用机制,研究NIK信号通路成为药物靶点的可能性; 2)研究lncRNA和RNA修饰在NIK信号调控中的作用机制; 3)研究lncRNA和RNA修饰在肥胖和糖尿病中的作用和调控机制。""近期论文
METTL3 is required for maintaining β-cell functionActivation of NF-κB-inducing kinase in islet β cells causes β-cell failure and diabetesMETTL3 is essential for postnatal development of brown adipose tissue and energy expenditure in miceDiscovery of a potent and selective NF-κB-inducing kinase (NIK) inhibitor that has anti-inflammatory effects in vitro and in vivoPurb promotes hepatic glucose production by increasing Adcy6 transcriptionIslet α-cell Inflammation Induced By NF-κB inducing kinase (NIK) Leads to Hypoglycemia, Pancreatitis, Growth Retardation, and Postnatal Death in MiceA small-molecule inhibitor of NFkB-inducing kinase (NIK) protects liver from toxin-induced inflammation, oxidative stress, and injury.Adapter proteins regulate insulin resistance and lipid metabolism in obesityA long non-coding RNA, APOA4-AS, regulates APOA4 expression depending on HuR in miceProgress and prospects of long noncoding RNAs in lipid homeostasis 相关热点